Saturday, March 1, 2025

ECG Blog #471 — Two for One?


The ECG in Figure-1 was obtained from a man in his 60s — who presented with an acute, febrile pulmonary illness. He has been short of breath — but not having chest pain.

QUESTION:
  • How would you interpret the ECG in Figure-1?

Figure-1: The initial ECG in today's case — obtained from a man in his 60s with dyspnea, but no chest pain. (To improve visualization — I've digitized the original ECG using PMcardio).

MY Thoughts on the ECG in Figure-1:
Interpretation of the ECG in Figure-1 is made more challenging by overlap of QRS complexes in multiple leads. That said — there appear to be 2 princpal elements to this rhythm: 
  • i) The run of wide beats that begins this ECG (Seen for beats #1-thru-8 — and then for two 3-beat salvos that are seen later in the tracing); and
  • ii) The lack of sinus P waves for the 2 short periods of narrower beats (ie, for beats #9-11; and 15-17).

PEARL: This is not an easy tracing to interpret. That said — When faced with a complex arrhythmia to interpret, in which there are a number of challenging elements — I find it helpful to: 
  • i) Look for an underlying rhythm.
  • ii) Begin with those elements that are easier to interpret.

I highlight these points in Figure-2:
  • Instead of sinus P waves — Don't the regularly-occurring RED lines in Figure-2 suggest the "sawtooth" pattern of AFlutter (Atrial Flutter) as the underlying rhythm? 
  • The rate of these RED lines is approximately 1 large box in duration — or close to 300/minute (which is perfectly consistent with the atrial rate of untreated AFlutter).
  • The R-R interval for these 6 supraventricular beats (ie, Narrow QRS complexes #9,10,11 and #15,16,17) — is close to 2 large boxes in duration, which is perfectly consistent to AFlutter with 2:1 AV conduction as the underlying rhythm.

This leaves us to contemplate the etiology of the wide beats:
  • As noted earlier — ECG #1 begins with a run of 8 wide beats (beats #1-thru-8 in Figure-2) — with two additional 3-beat runs (beats #9-11 and 15-17). Determining whether these wide beats represent ventricular beats vs supraventricular beats with aberrant conduction is the more difficult part of this tracing.

Figure-2: I've labeled flutter waves — here best seen in lead III.


Assessing the Wide Beats:
Overall — the wide beats appear to be fairly (but not completely) regular. This slight irregularity is best appreciated by looking at the R-R interval in lead II of Figure-3 between beats #12-13 vs between beats #13-14.
  • The overall rate of the wide beats is a bit over 150/minute (ie, the R-R interval is less than 2 large boxes in duration).
  • In contrast — I thought the narrow beats during AFlutter manifested a more consistently regular rate (as is common with 2:1 AFlutter).
  • There are 2 post-ectopic pauses in Figure-3 (highlighted by the double YELLOW arrows). Post-ectopic pauses are a common feature following a run of VT (Ventricular Tachycardia).
  • Doesn't the QRS complex of beat #9 in several leads (especially in leads II,III,aVF) look intermediate in morphology between the wide beats that precede it and narrow beats #10,11 that come after it? I therefore thought beat #9 was a fusion beat ("F" in Figure-3) — which if correct, would provide support that the wide beats in today's rhythm were ventricular.
  • Finally — QRS morphology of the wide beats in Figure-3 strongly suggests a ventricular etiology because: i) Transition in the chest leads occurs much earlier than expected for Left Bundle Branch Block conduction (ie, the R wave is already all positive by lead V4 — whereas transition usually does not occur with LBBB until at least lead V5, if not V6); ii) LBBB conduction typically manifests a wider monophasic R wave in lead I than in leads II and III — and — LBBB conduction does not produce an all negative QRS in lead aVL (as we see here); — and, iii) The frontal plane axis of the wide beats in Figure-3 is vertical (ie, most positive in leads II,III,aVF compared to lead I ) — which results in a QRS morphology consistent with RVOT VT (Right Ventricular Outflow Track VT) — namely, LBBB-like morphology in the chest leads with an inferior frontal plane axis.
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  • NOTE: See ECG Blog #204 (for more on "typical" QRS morphology with LBBB & RBBB, including an ECG Video) — and ECG Blog #323 (for more on the appearance of idiopathic VT, including RVOT VT which is the most common form).

Figure-3: I've labeled post-ectopic pauses and a probable fusion beat.


BOTTOM Line: It's impossible to be 100% certain as to the etiology of the wide beats in today's tracing. That said:
  • The underlying rhythm appears to be AFlutter with 2:1 AV conduction.
  • The ECG features described above make it most likely that the runs of wide beats in today's rhythm represent NSVT (Non-Sustained Ventricular Tachycardia). We don't know how long the 1st run is (since the tracing begins with VT). After a brief pause — we then see two 3-beat salvos.
  • Without more information — it's hard to know what optimal treatment should be. Both AFlutter and PVCs (including NSVT) are commonly seen with hypoxemia — and often resolve once the pulmonary problem and oxygen status are stabilized. Cardioversion is not indicated — since the runs of VT are not sustained. More information is needed.
  • Final Thought: I generally look for a single explanation to most of the arrhythmias I encounter. That said — today's case provides an insightful example of ECG features that suggest the occurrence of both an underlying supraventricular rhythm ( = AFlutter) — and, a superimposed ventricular rhythm (ie, repetitive PVCs with a run of NSVT).

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Acknowledgment: My appreciation for the anonymous donation of today's case and this tracing.
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Additional Relevant ECG Blog Posts to Today’s Case:

  • ECG Blog #185 — Reviews my System for Rhythm Interpretation — with use of the Ps, Qs & 3R Approach.

  • ECG Blog #287 — Review of AFlutter ...

  • ECG Blog #220 — Review of the approach to the regular WCT ( = Wide-Complex Tachycardia).
  • ECG Blog #196 — Another Case with a regular WCT.
  • ECG Blog #263 and Blog #283 — Blog #361 — Blog #384 — Blog #460 — and Blog #468 More WCT Rhythms ...

  • ECG Blog #197 — Reviews the concept of Idiopathic VT, of which Fascicular VT is one of the 2 most common types. 

  • ECG Blog #204 — Reviews the ECG diagnosis of the Bundle Branch Blocks (RBBB/LBBB/IVCD). 
  • ECG Blog #203 — Reviews ECG diagnosis of Axis and the Hemiblocks. For review of QRS morphology with the Bifascicular Blocks (RBBB/LAHB; RBBB/LPHB) — See the Video Pearl in this blog post.

  • ECG Blog #211 — WHY does Aberrant Conduction occur?
  • ECG Blog #301 — Reviews a WCT that is SupraVentricular! (with LOTS on Aberrant Conduction).
  • ECG Blog #445 and Blog #361 — Another regular WCT rhythm ...

  • ECG Blog #323 — Review of Fascicular VT.
  • ECG Blog #38 and Blog #85 — Review of Fascicular VT.
  • ECG Blog #278 — Another case of a regular WCT rhythm in a younger adult.
  • ECG Blog #35 — Review of RVOT VT
  • ECG Blog #42 — Criteria to distinguish VT vs Aberration.














4 comments:

  1. Thanks for this case! I don't understand why it can't be aberrantly conducted flutter. Could you elaborate on this?

    ReplyDelete
    Replies
    1. Thanks for your question. Please go back and review again my discussion under "Assessing the Wide Beats". This is not 100% — but I suggest there are multiple reasons why it is much more likely for the wide beats to be ventricular.

      Remember — Ventricular beats in this setting with this particular QRS morphology are much more common than aberrant conduction. You have to prove aberrancy — rather than the other way around — and I see no compelling reasons for aberrant conduction (ie, these are relatively late-cycle beats without any resemblance to underlying QRS morphology ... ). I hope this is helpful — :)

      Delete
  2. Replies
    1. I acknowledge that the "linking phenomenon" extends beyond my expertise.
      = = = = =
      A quick definition regarding linking — "In ECG (electrocardiogram) interpretation, "linking" refers to a phenomenon where a premature beat (e.g., a beat originating from an abnormal pathway) can prolong the refractoriness (recovery time) of the right bundle branch, potentially causing a functional right bundle branch block in the subsequent normal beat. Type 1 linking is characterized by a wide QRS complex following the premature beat, despite a relatively long R-R interval. This phenomenon is particularly relevant in the context of functional bundle branch blocks, especially in cases of intraventricular conduction delays."
      = = = = =
      In today's case, I discuss all the reasons I feel the wide beats are ventricular under, "Assessing the Wide Beats". In particular — I thought QRS morphology fits perfectly for RVOT VT ...

      Delete

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